Annual Review of Immunology Volume 26 2008 by Annual Reviews

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For example, Graves’ disease will debut at a younger age in T1858/T1858 homozygous patients compared with patients with C1858/C1858 genotype, whereas heterozygous patients develop the disease at an intermediate age (72). In contrast, no correlation between the T allele and severity of Graves’ disease has been demonstrated (72). Likewise, the age at onset for T1D is lower for patients with the T1858/T1858 genotype compared with the C1858/C1858 genotype, but no difference has been seen with regard to autoantibodies (38, 73).

19:812–17 130. Daha MR, Austen KF, Fearon DT. 1978. Heterogeneity, polypeptide composition and antigenic reactivity of C3NeF. J. Immunol. 120:1389–94 131. Austen KF. 2004. Acceptance of the Kober Medal: It only gets better. J. Clin. Invest. 114:1177 28 Austen ANRV338-IY26-02 ARI 16 February 2008 11:8 Annu. Rev. Immunol. 26:29-55. org by Shanghai Information Center for Life Sciences on 04/28/08. For personal use only. Protein Tyrosine Phosphatases in Autoimmunity Torkel Vang, Ana V. Miletic, Yutaka Arimura, Lutz Tautz, Robert C.

Another explanation for the augmented inhibitory effect of LYP∗ W620 on TCR signaling is altered subcellular localization, which may be tightly connected with a new set of interacting partners for LYP∗ W620 compared with LYP∗ R620. There appear to be subtle differences between the two LYP variants as evaluated by subcellular localization analyses and biochemical fractionation studies (T. V. Miletic & T. Mustelin, unpublished data), but more studies are required to clarify these issues. A final explanation for the increased inhibitory potential of LYP∗ W620 is an alteration in the substrate specificities and/or substrate affinities compared with LYP∗ R620, although this has yet to be experimentally tested.

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