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Even if the application of human antibodies as scientific therapeutics for melanoma and immune ailments has been well-established, it's only commencing to be learned for the therapy of viral infectious ailments. Polyclonal immunoglobulins have lengthy been used for a few viral ailments, yet they've got restricted efficiency and affliction scope.
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Extra info for Annual Review of Immunology Volume 5 1987
R. 1972. Isolation and characterization of Clq, a subcomponent of the first component of complement, from humanand rabbit sera. Biochem. J. 130:749-63 17. Schumaker, V. , Calcott, M. , Spiegelberg, H. , Muller-Eberhard, H. J. 1976. Ultracentrifuge studies of the binding of IgG of different subclasses to the Clq subunit of the first component of complement. Biochemistry 15:5175-81 18. Liberti, P. , Paul, S. M. 1978. Gross conformation of Clq: A subcomponent of the first componentof complement. Biochemistry 17:1952-58 19.
Arlaud, G. , Gagnon, J. 1985. Identification of the peptide bond cleaved during activation of humanC 1 r. FEBS Lett. 180:234-38 30. Arlaud, G. , Gagnon, J. 1983. Complete amino acid sequence of the catalytic chain of humancomplement subcomponent CYr. Biochemistry 22 : 1758-64 31. Villiers, C. , Arlaud, G. , Colomb, M. G. 1984. Autoactivation of human subcomponent Clr involves structural changes reflected in modifications of intrinsic fluorescence, circular dichroism and reactivity with monoclonal antibodies.
1982. Dynamicaspects of anti- Annual Reviews 42 81. Annu. Rev. Immunol. 5:21-42. org by HINARI on 08/28/07. For personal use only. 82. 83. 84. 85. 86. 87. 88. 89. 90. 91. SCHUMAKER~ ZAVODSZKY & POON body function. In The Antigens, ed. M. Sela, 6: 1~8. New York: Academic Press. 453 pp. , Jaton, J. C. 1980. Antigen-independent activation of the first component of complement C1 by chemically crosslinked rabbit IgG oligomers. FEBS Lett. , Kazatchkine, M. , Duc, H. , Liacopoulos, P. 1985. Activation of the human classical complement pathway by a mouse monoclonal hybrid IgG1-2a monovalent anti-TNP antibody bound to TNP-conjugated cells.